In normal breathing, air is sucked through >> << bronchial tract and down into finer
network of tubes in the lungs is called
, which many millions of tiny sacs surrounded by capillaries. They absorb the
and pass it. When toxins such as
smoke when inhaled into the lungs, particles caught in the trap and get
localized response. Chemicals that are allocated during the inflammatory response (eg,
) can destroy the walls of the alveoli >> << (). This is an important pathogenic event (break wall) results
significant, purely mechanical modifications easily
architecture due to lung elastic recoil which causes parenchyma collapse
partitions divide the region and holds vnutrilehochnoho air
collection that exceeds the area of the alveoli, made in connection >> << to break walls.
Since the process is the expansion of the chest (barrel chest
) and diaphragm contraction (flattening) is an increase in inspiratory
reexpansion / ventilation residual partially collapsed healthy
parenchyma. In addition lasix drug dose, the more
depends on the chest and abdominal muscle action,
particular, the end expiratory phase, and not that
physiological impact of lung elastic, thus provoking a bronchial >> << obstruction. Decreased mainly the ability to provide carbon dioxide
due to lack of ventilation, and in more serious cases,
oxygen also violated. The activity of another molecule called
alpha-1-antitrypsin normally neutralizes the destructive action >> << one of these damaging molecules. .
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